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当使用该小鼠品系发表文献时,请引用原始文献,并在材料方法中提供该品系的品系货号:JAX stock#001976Therapeutic administration of Tregitope-Human Albumin Fusion with Insulin Peptides to promote Antigen-Specific Adaptive Tolerance Induction.
De Groot AS , et al.
Complete overview of protein-inactivating sequence variations in 36 sequenced mouse inbred strains.
Timmermans S , et al.
Genetic Analysis of Substrain Divergence in Non-Obese Diabetic (NOD) Mice.
Simecek P , et al.
Initiation of autoimmune diabetes in NOD/Lt mice is MHC class I-dependent.
Serreze DV , et al.
The NOD mouse: A model for analyzing the interplay between heredity and environment in development of autoimmune disease.
Leiter EH , et al.
Breeding of a non-obese, diabetic strain of mice.
Makino S , et al.
Unstable FoxP3+ T regulatory cells in NZW mice.
Depis F , et al.
Mouse genomic variation and its effect on phenotypes and gene regulation.
Keane TM , et al.
Social approach and repetitive behavior in eleven inbred mouse strains.
Moy SS , et al.
Development of a SNP genotyping panel for genetic monitoring of the laboratory mouse.
Petkov PM , et al.
Strain distribution pattern of susceptibility to immune-mediated nephritis.
Xie C , et al.
Adenovirus early region 3 antiapoptotic 10.4K, 14.5K, and 14.7K genes decrease the incidence of autoimmune diabetes in NOD mice.
Pierce MA , et al.
During the early prediabetic period in NOD mice, the pathogenic CD8(+) T-cell population comprises multiple antigenic specificities.
DiLorenzo TP , et al.
Cytotoxic T lymphocyte and cDNA sequence analyses of the MHC class Ib molecule Qa1 in nonobese diabetic mice.
Chun T , et al.
Reduced oral wound healing in the NOD mouse model for type 1 autoimmune diabetes and its reversal by epidermal growth factor supplementation.
Nagy A , et al.
Autoantibody Patterns in Diabetes-prone NOD Mice and in Standard C57BL/6 Mice.
Quintana FJ , et al.
Il-12 administration reveals diabetogenic t cells in genetically resistant i-ealpha-transgenic nonobese diabetic mice: resistance to autoimmune diabetes is associated with binding of ealpha-derived peptides to the i-a(g7) molecule.
Trembleau S , et al.
Interferon-gamma receptor signaling is dispensable in the development of autoimmune type 1 diabetes in NOD mice.
Serreze DV , et al.
NOD Mice and Related Strains: Origins, Husbandry and Biology Introduction
Leiter EH , et al.
Apoptosis is increased in a model of diabetes-impaired wound healing in genetically diabetic mice.
Darby IA , et al.
Studies on the polymorphism of the fifth component of complement in laboratory mice.
Lynch DM , et al.
Prevention of diabetes in the NOD mouse: implications for therapeutic intervention in human disease.
Bowman MA , et al.
Genetic and pathogenic basis of autoimmune diabetes in NOD mice.
Serreze DV , et al.
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